Front | Back |
Muscle fibers
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Small number of motor units are involuntarily activated to produce a sustained contraction of their mm fibers
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During a muscle contraction
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The I bands and H zones become narrower and eventually disappear when the mm is maximally contracted
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Acetylcholinesterase
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Breaks down ACH at the synapse causes muscle relaxation
(mm fiber will not get excited) |
Acetylcholinesterase inhibitors
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Slow the removal of ACh enhances mm contraction
used in treating alzheimer's patients |
Botulinum toxin
|
Stops ACh from being barfed into the synapse - causing diaphragm paralysis and is found in improperly canned food
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Curare
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Blocks ACh from pushing the doorbell
natives used as a poison in arrow darts used in surgery to relay skeletal mm |
Rigor mortis
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Calcium leaks out after death stimulating muscles to contract
|
Slow oxidative (type I) mm
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Very fatigue resistant
back and neck mm need O2 at a slow and steady rate postural mm |
Fast oxidative (type IIa) mm
FOG |
Moderate fatigue resistance
lactic acid build up over time leg mm needs O2 at a faster rate for faster contraction |
Fast glycolytic (type IIb) mm
|
Fatigue quickly
most powerful contraction, most rapid arm and shoulder mm no O2 is used (anaerobic process) |
Autorhythmicity
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Built in intrinsic contractions
does not need ACh to stimulate cardiac and smooth mm |
Hypertrophy
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Increase mm diameter
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2 degree sprain
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Partially torn ligament
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Strain
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Overstretching of mm or tendon
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Muscle spasm or cramp
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Causes: electrolyte imbalance, ischemia, exercise induced, splinting
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