Circle of Willis, Brain- Associated Area/deficit

Contralateral hemiparesis (lower extremities), medial leminiscus (decrease in contralateral proprioception), ipsilateral paralysis of hypoglossal nerve.

Contralateral loss of pain and temperature, ipsilateral dysphagia, hoarseness, decrease in gag reflex, vertigo, diplopoia, nystagmus, vomiting, ipsilateral Horner's, ipsilateral facial pain and temperature, trigeminal nucleus (spinal tract and nucleus), ipsilateral ataxia.

Ipsilateral facial paralysis; ipsilateral cochlear nucleus, vestibular (nystagmus), ipsilateral facial pain and temperature, ipsilateral dystaxia (MCP, ICP)

Contralateral hemianopia with macular sparing, supplies occipital cortex

Contralateral face and arm paralysis and sensory loss, aphasia (dominant sphere), left-sided neglect.

Supplies medial surface of the brain, leg-foot area of motor and sensory cortices.

Most common site of circle of Willis aneurysm; lesions may cause visual field defects.

Common area of aneurysm; causes CN III palsy.

Divisions of middle cerebral artery; supply internal capsule, caudate, putamen, globus pallidus. "Arteries of stroke"; infarct of the posterior limb of the internal capsule causes pure motor hemiparesis.

Between anterior cerebral/middle cerebral, posterior/middle cerebral arteries. Damage in severe hypotension -- > upper leg/upper arm weakness, defects in higher-order visual processing.

Infarct causes "locked in syndrome" (CN III is typically intact)

General sensory and motor dysfunction, aphasia.

Cranial nerve deficits (vertigo, visual deficits), coma, cerebellar deficits (ataxia). Dominant hemisphere (ataxia), non-dominant (neglect).